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Results by year

Year	Number of Results
2002	6
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2012	118
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1,497 results

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Page 1

[leukocyte-associated immunoglobulin-like receptor 1 (LAIR-1) inhibits proliferation and promotes apoptosis of human HEL cells with JAK2 V617F mutation by blocking the JAK/STAT and PI3K/AKT signaling pathways].

Fan C, Zhang Y, Yang R, Wu X, Zhou J, Xue J. Fan C, et al. Xi Bao Yu Fen Zi Mian Yi Xue Za Zhi. 2024 Mar;40(3):207-214. Xi Bao Yu Fen Zi Mian Yi Xue Za Zhi. 2024. PMID: 38512030 Chinese.

Molecular basis of JAK2 activation in erythropoietin receptor and pathogenic JAK2 signaling.

Abraham BG, Haikarainen T, Vuorio J, Girych M, Virtanen AT, Kurttila A, Karathanasis C, Heilemann M, Sharma V, Vattulainen I, Silvennoinen O. Abraham BG, et al. Sci Adv. 2024 Mar 8;10(10):eadl2097. doi: 10.1126/sciadv.adl2097. Epub 2024 Mar 8. Sci Adv. 2024. PMID: 38457493 Free PMC article.

Impact of CALR and JAK2V617F Mutations on Clinical Course and Disease Outcomes in Essential Thrombocythemia: A Multicenter Retrospective Study in Turkish Patients.

Narlı Özdemir Z, İpek Y, Patır P, Ermiş G, Çiftçiler R, Özmen D, Baysal M, Gürsoy V, Yıldızhan E, Güven S, Ercan T, Elibol T, Mersin S, Genç E, Davulcu EA, Karakuş V, Erkut N, Güneş G, Diz Küçükkaya R, Eşkazan AE. Narlı Özdemir Z, et al. Turk J Haematol. 2024 Mar 1;41(1):26-36. doi: 10.4274/tjh.galenos.2024.2023.0430. Turk J Haematol. 2024. PMID: 38433449 Free PMC article.

Knockdown of liver cancer cell-secreted exosomal PSMA5 controls macrophage polarization to restrain cancer progression by blocking JAK2/STAT3 signaling.

Xie S, Li X, Yan J, Yu H, Chen S, Chen K. Xie S, et al. Immun Inflamm Dis. 2024 Feb;12(2):e1146. doi: 10.1002/iid3.1146. Immun Inflamm Dis. 2024. PMID: 38415977 Free PMC article.

Sciellin promotes the development and progression of thyroid cancer through the JAK2/STAT3 signaling pathway.

Guo H, Wang Z, Yin K, Ma R, Zhang Y, Yin F, Li H, Yin D. Guo H, et al. Mol Carcinog. 2024 Apr;63(4):701-713. doi: 10.1002/mc.23682. Epub 2024 Feb 27. Mol Carcinog. 2024. PMID: 38411346

[Comparison of Clinical Characteristics of JAK2, CALR and Tri-Negative Driving Mutant Type in Patients with Essential Thrombocythemia].

Li YM, Yang EP, Wang ZQ, Wang DH, Niu JC, Li YJ, Ming J, Sun MQ, Chen Z, Liu WY, Lyu Y, Hu XM. Li YM, et al. Zhongguo Shi Yan Xue Ye Xue Za Zhi. 2024 Feb;32(1):197-201. doi: 10.19746/j.cnki.issn.1009-2137.2024.01.031. Zhongguo Shi Yan Xue Ye Xue Za Zhi. 2024. PMID: 38387921 Chinese.

Targeting C21orf58 is a Novel Treatment Strategy of Hepatocellular Carcinoma by Disrupting the Formation of JAK2/C21orf58/STAT3 Complex.

Jiang H, Wang Y, Wen D, Yu R, Esa SS, Lv K, Feng Q, Liu J, Li F, He L, Di X, Zhang S. Jiang H, et al. Adv Sci (Weinh). 2024 Apr;11(15):e2306623. doi: 10.1002/advs.202306623. Epub 2024 Feb 11. Adv Sci (Weinh). 2024. PMID: 38342622 Free PMC article.

Altered erythropoiesis via JAK2 and ASXL1 mutations in myeloproliferative neoplasms.

Collins TB, Laranjeira ABA, Kong T, Fulbright MC, Fisher DAC, Sturgeon CM, Batista LFZ, Oh ST. Collins TB, et al. Exp Hematol. 2024 Apr;132:104178. doi: 10.1016/j.exphem.2024.104178. Epub 2024 Feb 9. Exp Hematol. 2024. PMID: 38340948

Calreticulin and JAK2V617F driver mutations induce distinct mitotic defects in myeloproliferative neoplasms.

Holl K, Chatain N, Krapp S, Baumeister J, Maié T, Schmitz S, Scheufen A, Brock N, Koschmieder S, Moreno-Andrés D. Holl K, et al. Sci Rep. 2024 Feb 2;14(1):2810. doi: 10.1038/s41598-024-53240-8. Sci Rep. 2024. PMID: 38308077 Free PMC article.

Proinflammatory phenotype of iPS cell-derived JAK2 V617F megakaryocytes induces fibrosis in 3D in vitro bone marrow niche.

Flosdorf N, Böhnke J, de Toledo MAS, Lutterbach N, Lerma VG, Graßhoff M, Olschok K, Gupta S, Tharmapalan V, Schmitz S, Götz K, Schüler HM, Maurer A, Sontag S, Küstermann C, Seré K, Wagner W, Costa IG, Brümmendorf TH, Koschmieder S, Chatain N, Castilho M, Schneider RK, Zenke M. Flosdorf N, et al. Stem Cell Reports. 2024 Feb 13;19(2):224-238. doi: 10.1016/j.stemcr.2023.12.011. Epub 2024 Jan 25. Stem Cell Reports. 2024. PMID: 38278152 Free PMC article.

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