PubMed for id: 23397666

Year	Number of Results
1995	3
1997	2
2022	4
2023	2
2024	0

1

Coordination of cross-talk between metabolism and epigenetic regulation by the SIN3 complex.

Soukar I, Amarasinghe A, Pile LA. Soukar I, et al. Enzymes. 2023;53:33-68. doi: 10.1016/bs.enz.2023.06.001. Epub 2023 Jul 28. Enzymes. 2023. PMID: 37748836 Review.

2

DNA methylation episignature for Witteveen-Kolk syndrome due to SIN3A haploinsufficiency.

Coenen-van der Spek J, Relator R, Kerkhof J, McConkey H, Levy MA, Tedder ML, Louie RJ, Fletcher RS, Moore HW, Childers A, Farrelly ER, Champaigne NL, Lyons MJ, Everman DB, Rogers RC, Skinner SA, Renck A, Matalon DR, Dills SK, Monteleone B, Demirdas S, Dingemans AJM, Donker Kaat L, Kolk SM, Pfundt R, Rump P, Sadikovic B, Kleefstra T, Butler KM. Coenen-van der Spek J, et al. Genet Med. 2023 Jan;25(1):63-75. doi: 10.1016/j.gim.2022.10.004. Epub 2022 Nov 18. Genet Med. 2023. PMID: 36399132 Free article.

3

The Sin3A/MAD1 Complex, through Its PAH2 Domain, Acts as a Second Repressor of Retinoic Acid Receptor Beta Expression in Breast Cancer Cells.

Dahiya NR, Leibovitch BA, Kadamb R, Bansal N, Waxman S. Dahiya NR, et al. Cells. 2022 Mar 31;11(7):1179. doi: 10.3390/cells11071179. Cells. 2022. PMID: 35406744 Free PMC article.

4

Suppression of SIN3A by miR-183 Promotes Breast Cancer Metastasis.

Davenport ML, Davis MR, Davenport BN, Crossman DK, Hall A, Pike J, Harada S, Hurst DR, Edmonds MD. Davenport ML, et al. Mol Cancer Res. 2022 Jun 3;20(6):883-894. doi: 10.1158/1541-7786.MCR-21-0508. Mol Cancer Res. 2022. PMID: 35247910 Free PMC article.

5

Proteome-scale mapping of binding sites in the unstructured regions of the human proteome.

Benz C, Ali M, Krystkowiak I, Simonetti L, Sayadi A, Mihalic F, Kliche J, Andersson E, Jemth P, Davey NE, Ivarsson Y. Benz C, et al. Mol Syst Biol. 2022 Jan;18(1):e10584. doi: 10.15252/msb.202110584. Mol Syst Biol. 2022. PMID: 35044719 Free PMC article.

6

Histone deacetylases associated with the mSin3 corepressor mediate mad transcriptional repression.

Laherty CD, Yang WM, Sun JM, Davie JR, Seto E, Eisenman RN. Laherty CD, et al. Cell. 1997 May 2;89(3):349-56. doi: 10.1016/s0092-8674(00)80215-9. Cell. 1997. PMID: 9150134 Free article.

7

Histone deacetylase activity is required for full transcriptional repression by mSin3A.

Hassig CA, Fleischer TC, Billin AN, Schreiber SL, Ayer DE. Hassig CA, et al. Cell. 1997 May 2;89(3):341-7. doi: 10.1016/s0092-8674(00)80214-7. Cell. 1997. PMID: 9150133 Free article.

8

Mad3 and Mad4: novel Max-interacting transcriptional repressors that suppress c-myc dependent transformation and are expressed during neural and epidermal differentiation.

Hurlin PJ, Quéva C, Koskinen PJ, Steingrímsson E, Ayer DE, Copeland NG, Jenkins NA, Eisenman RN. Hurlin PJ, et al. EMBO J. 1995 Nov 15;14(22):5646-59. doi: 10.1002/j.1460-2075.1995.tb00252.x. EMBO J. 1995. PMID: 8521822 Free PMC article.

9

Mad-Max transcriptional repression is mediated by ternary complex formation with mammalian homologs of yeast repressor Sin3.

Ayer DE, Lawrence QA, Eisenman RN. Ayer DE, et al. Cell. 1995 Mar 10;80(5):767-76. doi: 10.1016/0092-8674(95)90355-0. Cell. 1995. PMID: 7889570 Free article.

10

A widely distributed putative mammalian transcriptional regulator containing multiple paired amphipathic helices, with similarity to yeast SIN3.

Halleck MS, Pownall S, Harder KW, Duncan AM, Jirik FR, Schlegel RA. Halleck MS, et al. Genomics. 1995 Mar 20;26(2):403-6. doi: 10.1016/0888-7543(95)80229-f. Genomics. 1995. PMID: 7601471

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