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Year | Number of Results |
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2002 | 1 |
2003 | 1 |
2009 | 1 |
2015 | 1 |
2024 | 0 |
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Early embryonic death of glutamate carboxypeptidase II (NAALADase) homozygous mutants.
Synapse. 2003 Dec 15;50(4):285-92. doi: 10.1002/syn.10263.
Synapse. 2003.
PMID: 14556233
Phenotypic characterization of mice heterozygous for a null mutation of glutamate carboxypeptidase II.
Han L, Picker JD, Schaevitz LR, Tsai G, Feng J, Jiang Z, Chu HC, Basu AC, Berger-Sweeney J, Coyle JT.
Han L, et al.
Synapse. 2009 Aug;63(8):625-35. doi: 10.1002/syn.20649.
Synapse. 2009.
PMID: 19347959
Free PMC article.
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Mice lacking glutamate carboxypeptidase II develop normally, but are less susceptible to traumatic brain injury.
Gao Y, Xu S, Cui Z, Zhang M, Lin Y, Cai L, Wang Z, Luo X, Zheng Y, Wang Y, Luo Q, Jiang J, Neale JH, Zhong C.
Gao Y, et al.
J Neurochem. 2015 Jul;134(2):340-53. doi: 10.1111/jnc.13123. Epub 2015 Apr 28.
J Neurochem. 2015.
PMID: 25872793
Free article.
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Deletion of the glutamate carboxypeptidase II gene in mice reveals a second enzyme activity that hydrolyzes N-acetylaspartylglutamate.
Bacich DJ, Ramadan E, O'Keefe DS, Bukhari N, Wegorzewska I, Ojeifo O, Olszewski R, Wrenn CC, Bzdega T, Wroblewska B, Heston WD, Neale JH.
Bacich DJ, et al.
J Neurochem. 2002 Oct;83(1):20-9. doi: 10.1046/j.1471-4159.2002.01117.x.
J Neurochem. 2002.
PMID: 12358725
Free article.
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