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Items: 1 to 20 of 37

1.
Oxytocin receptors in the Magel2 mouse model of autism: Specific region, age, sex and oxytocin treatment effects
Valentina Gigliucci, Marta Busnelli, Francesca Santini, Camilla Paolini, Alessandra Bertoni, Fabienne Schaller, Françoise Muscatelli, Bice Chini
Front Neurosci. 2023; 17: 1026939. Published online 2023 Mar 14. doi: 10.3389/fnins.2023.1026939
PMCID:
PMC10043208
AbstractArticlePDF–2.7M
2.
Deficiency of the paternally inherited gene Magel2 alters the development of separation‐induced vocalization and maternal behavior in mice
Gabriela M. Bosque Ortiz, Gustavo M. Santana, Marcelo O. Dietrich
Genes Brain Behav. 2022 Jan; 21(1): e12776. Published online 2021 Nov 23. doi: 10.1111/gbb.12776
PMCID:
PMC9744533
AbstractArticlePDF–2.8M
3.
Early life oxytocin treatment improves thermo-sensory reactivity and maternal behavior in neonates lacking the autism-associated gene Magel2
Laura Caccialupi Da Prato, Ugo Zayan, Dina Abdallah, Vanessa Point, Fabienne Schaller, Emilie Pallesi-Pocachard, Aurélie Montheil, Stéphane Canaan, Jean-Luc Gaiarsa, Françoise Muscatelli, Valéry Matarazzo
Neuropsychopharmacology. 2022 Oct; 47(11): 1901–1912. Published online 2022 Apr 8. doi: 10.1038/s41386-022-01313-5
PMCID:
PMC9485246
AbstractArticlePDF–2.2M
4.
The Metabolic Efficacy of a Cannabidiolic Acid (CBDA) Derivative in Treating Diet- and Genetic-Induced Obesity
Elad Ben-Cnaan, Anna Permyakova, Shahar Azar, Shira Hirsch, Saja Baraghithy, Liad Hinden, Joseph Tam
Int J Mol Sci. 2022 May; 23(10): 5610. Published online 2022 May 17. doi: 10.3390/ijms23105610
PMCID:
PMC9144717
AbstractArticlePDF–3.6M
5.
Complex Economic Behavior Patterns Are Constructed from Finite, Genetically Controlled Modules of Behavior
Cornelia N. Stacher Hörndli, Eleanor Wong, Elliott Ferris, Kathleen Bennett, Susan Steinwand, Alexis Nikole Rhodes, P. Thomas Fletcher, Christopher Gregg
Cell Rep. Author manuscript; available in PMC 2020 Sep 7.
Published in final edited form as: Cell Rep. 2019 Aug 13; 28(7): 1814–1829.e6. doi: 10.1016/j.celrep.2019.07.038
PMCID:
PMC7476553
AbstractArticlePDF–2.6M
6.
Two mouse models carrying truncating mutations in Magel2 show distinct phenotypes
Daisuke Ieda, Yutaka Negishi, Tomomi Miyamoto, Yoshikazu Johmura, Natsuko Kumamoto, Kohji Kato, Ichiro Miyoshi, Makoto Nakanishi, Shinya Ugawa, Hisashi Oishi, Shinji Saitoh
PLoS One. 2020; 15(8): e0237814. Published online 2020 Aug 17. doi: 10.1371/journal.pone.0237814
PMCID:
PMC7430741
AbstractArticlePDF–2.0M
7.
mTOR and autophagy pathways are dysregulated in murine and human models of Schaaf-Yang syndrome
Emeline Crutcher, Rituraj Pal, Fatemeh Naini, Ping Zhang, Magdalena Laugsch, Jean Kim, Aleksandar Bajic, Christian P. Schaaf
Sci Rep. 2019; 9: 15935. Published online 2019 Nov 4. doi: 10.1038/s41598-019-52287-2
PMCID:
PMC6828689
AbstractArticlePDF–4.1M
8.
Magel2 knockout mice manifest altered social phenotypes and a deficit in preference for social novelty
Michael D. Fountain, Huifang Tao, Chun-An Chen, Jiani Yin, Christian P. Schaaf
Genes Brain Behav. Author manuscript; available in PMC 2018 Jul 1.
Published in final edited form as: Genes Brain Behav. 2017 Jul; 16(6): 592–600. Published online 2017 Apr 4. doi: 10.1111/gbb.12378
PMCID:
PMC5495607
AbstractArticlePDF–688K
9.
Loss of Magel2 impairs the development of hypothalamic Anorexigenic circuits
Julien Maillard, Soyoung Park, Sophie Croizier, Charlotte Vanacker, Joshua H. Cook, Vincent Prevot, Maithe Tauber, Sebastien G. Bouret
Hum Mol Genet. 2016 Aug 1; 25(15): 3208–3215. Published online 2016 Jun 10. doi: 10.1093/hmg/ddw169
PMCID:
PMC5179922
AbstractArticlePDF–528K
10.
Impaired Hypothalamic Regulation of Endocrine Function and Delayed Counterregulatory Response to Hypoglycemia in Magel2-Null Mice
Alysa A. Tennese, Rachel Wevrick
Endocrinology. 2011 Mar; 152(3): 967–978. Published online 2011 Jan 19. doi: 10.1210/en.2010-0709
PMCID:
PMC3198964
AbstractArticle
11.
Loss of Magel2, a Candidate Gene for Features of Prader-Willi Syndrome, Impairs Reproductive Function in Mice
Rebecca E. Mercer, Rachel Wevrick
PLoS One. 2009; 4(1): e4291. Published online 2009 Jan 27. doi: 10.1371/journal.pone.0004291
PMCID:
PMC2627930
AbstractArticlePDF–710K
12.
Magel2 knockdown in hypothalamic POMC neurons innervating the medial amygdala reduces susceptibility to diet-induced obesity
Yuna Choi, Hyeon-Young Min, Jiyeon Hwang, Young-Hwan Jo
Life Sci Alliance. 2022 Nov; 5(11): e202201502. Published online 2022 Aug 25. doi: 10.26508/lsa.202201502
PMCID:
PMC9418835
AbstractArticlePDF–2.4M
13.
Loss of MAGEL2 in Prader-Willi syndrome leads to decreased secretory granule and neuropeptide production
Helen Chen, A. Kaitlyn Victor, Jonathon Klein, Klementina Fon Tacer, Derek J.C. Tai, Celine de Esch, Alexander Nuttle, Jamshid Temirov, Lisa C. Burnett, Michael Rosenbaum, Yiying Zhang, Li Ding, James J. Moresco, Jolene K. Diedrich, John R. Yates, III, Heather S. Tillman, Rudolph L. Leibel, Michael E. Talkowski, Daniel D. Billadeau, Lawrence T. Reiter, Patrick Ryan Potts
JCI Insight. 2020 Sep 3; 5(17): e138576. Published online 2020 Sep 3. doi: 10.1172/jci.insight.138576
PMCID:
PMC7526459
AbstractArticlePDF–12M
14.
A MAGEL2-deubiquitinase complex modulates the ubiquitination of circadian rhythm protein CRY1
K. Vanessa Carias, Mercedes Zoeteman, Abigail Seewald, Matthea R. Sanderson, Jocelyn M. Bischof, Rachel Wevrick
PLoS One. 2020; 15(4): e0230874. Published online 2020 Apr 21. doi: 10.1371/journal.pone.0230874
PMCID:
PMC7173924
AbstractArticlePDF–2.1M
15.
Magel2 Is Required for Leptin-Mediated Depolarization of POMC Neurons in the Hypothalamic Arcuate Nucleus in Mice
Rebecca E. Mercer, Sheldon D. Michaelson, Melissa J. S. Chee, Tanya A. Atallah, Rachel Wevrick, William F. Colmers
PLoS Genet. 2013 Jan; 9(1): e1003207. Published online 2013 Jan 17. doi: 10.1371/journal.pgen.1003207
PMCID:
PMC3547795
AbstractArticlePDF–1.0M
16.
Necdin, a Prader–Willi syndrome candidate gene, regulates gonadotropin-releasing hormone neurons during development
Nichol L.G. Miller, Rachel Wevrick, Pamela L. Mellon
Hum Mol Genet. 2009 Jan 15; 18(2): 248–260. Published online 2008 Oct 17. doi: 10.1093/hmg/ddn344
PMCID:
PMC2638776
AbstractArticlePDF–1006K
17.
The Prader-Willi syndrome murine imprinting center is not involved in the spatio-temporal transcriptional regulation of the Necdin gene
Françoise Watrin, Elodie Le Meur, Nathalie Roeckel, Marie-Anne Ripoche, Luisa Dandolo, Françoise Muscatelli
BMC Genet. 2005; 6: 1. Published online 2005 Jan 5. doi: 10.1186/1471-2156-6-1
PMCID:
PMC545062
AbstractArticlePDF–1.0M
18.
The Prader-Willi syndrome proteins MAGEL2 and necdin regulate leptin receptor cell surface abundance through ubiquitination pathways
Tishani Methsala Wijesuriya, Leentje De Ceuninck, Delphine Masschaele, Matthea R Sanderson, Karin Vanessa Carias, Jan Tavernier, Rachel Wevrick
Hum Mol Genet. 2017 Nov 1; 26(21): 4215–4230. Published online 2017 Aug 14. doi: 10.1093/hmg/ddx311
PMCID:
PMC5886282
AbstractArticlePDF–1.1M
19.
Magel2, a Prader-Willi syndrome candidate gene, modulates the activities of circadian rhythm proteins in cultured cells
Julia Devos, Sara V Weselake, Rachel Wevrick
J Circadian Rhythms. 2011; 9: 12. Published online 2011 Dec 30. doi: 10.1186/1740-3391-9-12
PMCID:
PMC3278377
AbstractArticlePDF–1.1M
20.
Necdin Promotes Ubiquitin-Dependent Degradation of PIAS1 SUMO E3 Ligase
Ibrahim Gur, Kazushiro Fujiwara, Koichi Hasegawa, Kazuaki Yoshikawa
PLoS One. 2014; 9(6): e99503. Published online 2014 Jun 9. doi: 10.1371/journal.pone.0099503
PMCID:
PMC4049815
AbstractArticlePDF–6.5M

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